Male-pattern baldness — androgenetic alopecia — is caused by a genetic sensitivity to DHT, a hormone that gradually shrinks vulnerable follicles until they stop producing visible hair. It typically starts at the temples and crown and progresses through the Norwood stages.
It rarely stops on its own. The hairline recedes, the crown thins, and over years the two zones can merge. The donor hair at the back and sides is DHT-resistant — which is exactly why it can be transplanted to permanently rebuild thinning areas.
Catching it early matters: every month of untreated loss miniaturizes more follicles you can't get back.
The right plan depends on your stage. Most men do best with a combination:
Male-pattern baldness is genetic sensitivity to DHT, a derivative of testosterone. In predisposed men, DHT gradually shrinks (“miniaturises”) follicles at the hairline and crown — each growth cycle produces a finer, shorter hair until the follicle stops. Crucially, the follicles at the back and sides are genetically DHT-resistant, which is why that band survives and why it makes the ideal permanent donor supply for a transplant.
Because the process is progressive, the goal is two-fold: slow the loss and restore what’s gone. Medication (finasteride, minoxidil) and PRP target the miniaturisation to protect native hair; an FUE transplant moves DHT-resistant follicles into the bald zones for a permanent result. Starting maintenance early preserves more of your own hair — and means any future transplant has less ground to cover.
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